PCS Thesis – UV Index 2

The prior post suggested the subject experienced a diminished exposure to solar UV radiation due to his maintaining a work schedule that precluded, or greatly reduced, his daily exposure to solar UV radiation.

The primary thesis being explored in this series of posts is the potential for a reduction in UV exposure to moderate, or significantly reduce, the ability of the pineal gland to engage in “producing sulfate, and that the sulfate, conjugated to melatonin, is distributed throughout the brain via the CSF [cerebral spinal fluid] .” Sulfate is understood to have a role in the creation of “structured water” which offers a critical neuro-protective benefit through “buffering the brain against physical impact, and in degrading and recycling damaged proteins and mitochondria.”

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PCS Thesis – UV Index

The prior post suggested that any mechanism which serves to diminish, or mitigate, the effects of mechanical insult to the brain will have profound benefit for the injury victim. Further, it was proposed that a deficiency in UV light exposure may impair the ability of the pineal gland to synthesize the required sulphate, and that it is this UV deficiency which serves to explain the increased severity of brain trauma expressed as PCS.

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PCS Thesis – The Pineal Gland

There exist two modes of injury in TBI. The first of these is the initial mechanical insult as described in the prior post and depicted in Figure 1 of that post. The second mode of injury, potentially the more severe, is the biochemical insult that follows subsequent to the mechanical insult and commences within a period of between 1 and 4 hours. This biochemical insult is triggered by the initial injury.
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