PCS Thesis – UV Index

The prior post suggested that any mechanism which serves to diminish, or mitigate, the effects of mechanical insult to the brain will have profound benefit for the injury victim. Further, it was proposed that a deficiency in UV light exposure may impair the ability of the pineal gland to synthesize the required sulphate, and that it is this UV deficiency which serves to explain the increased severity of brain trauma expressed as PCS.

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PCS Thesis – The Pineal Gland

There exist two modes of injury in TBI. The first of these is the initial mechanical insult as described in the prior post and depicted in Figure 1 of that post. The second mode of injury, potentially the more severe, is the biochemical insult that follows subsequent to the mechanical insult and commences within a period of between 1 and 4 hours. This biochemical insult is triggered by the initial injury.
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PCS Thesis – Injury Modalities

This post introduces two biological mechanisms believed to have contributed to the severity of the subject’s mTBI. On the balance of evidence, there is strong reason to believe that either, or both, of these injury modalities may play a significant role in the severity of mTBI injury, and therefore serve as contributing factors to the symptomology described as PCS.

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